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More pieces to the puzzle showing beneficial effects of exercise after myocardial infarction

Exercise Training Stabilizes RyR2-Dependent Ca2+ Release in Post-infarction Heart Failure.

Tore Kristian Danielsen, Mani Sadredini, Ravinea Manotheepan, Jan Magnus Aronsen, Michael Frisk, Marie Haugsten Hansen, Kjetil Wessel Andressen, Karina Hougen, Finn Olav Levy, William E. Louch, Ole Mathias Sejersted, Ivar Sjaastad and Mathis Korseberg Stokke

Exercise is good for the heart, and even more so after a myocardial infarction. But why? How does exercise affect the remaining working cells of the heart? Building on previous work by the Stokke Group, PhD student Tore Kristian Danielsen and colleagues explored how exercise affects cellular processes that can lead to arrhythmias. The results indicate a combination of several factors, comprising altered intracellular calcium handling and sympathetic signaling.

Read the article in Frontiers in Cardiovascular Medicine

doi: 10.3389/fcvm.2020.623922.

Tore Kristian Danielsen

Doctoral Research Fellow

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Mathis Korseberg Stokke

Head of Department & Group Leader & Professor & Senior Consultant

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Institute for Experimental Medical Research

Oslo University Hospital, Ullevål

PB 4956 Nydalen

NO-0424 Oslo

Norway

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