Tore Kristian Danielsen, Mani Sadredini, Ravinea Manotheepan, Jan Magnus Aronsen, Michael Frisk, Marie Haugsten Hansen, Kjetil Wessel Andressen, Karina Hougen, Finn Olav Levy, William E. Louch, Ole Mathias Sejersted, Ivar Sjaastad and Mathis Korseberg Stokke
Exercise is good for the heart, and even more so after a myocardial infarction. But why? How does exercise affect the remaining working cells of the heart? Building on previous work by the Stokke Group, PhD student Tore Kristian Danielsen and colleagues explored how exercise affects cellular processes that can lead to arrhythmias. The results indicate a combination of several factors, comprising altered intracellular calcium handling and sympathetic signaling.
Read the article in Frontiers in Cardiovascular Medicine
doi: 10.3389/fcvm.2020.623922.
