Image: Profile picture of Jia Li

Jia Li

Postdoctoral fellow

Louch group

Email: jia.li@medisin.uio.no

Education

PhD in Department of Medical Cell Biology, Medical Faculty, Uppsala University, Sweden

Positions

2018-2021 Postdoc researcher
Institute for Experimental Medical Research (IEMR), Ullevål Sykehus

2015-2018 Postdoc researcher
Institute for Experimental Medical Research (IEMR), Oslo University

Funding

2018 3 years postdoc fellow granted by Nasjonalforeningen

Academic interests

Cellular mechanism of Frank-Starling law in healthy and failing heart.

Research area

  • Cardiomyocyte signaling
  • Excitation contraction coupling
  • Heart failure
  • Imaging

Latest publications

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Ruud M, Frisk M, Melleby AO, Norseng PA, Mohamed BA, Li J, Aronsen JM, Setterberg IE, Jakubiczka J, van Hout I, Coffey S, Shen X, Nygård S, Lunde IG, Tønnessen T, Jones PP, Sjaastad I, Gullestad L, Toischer K, Dahl CP, Christensen G, Louch WE (2024)
Regulation of cardiomyocyte t-tubule structure by preload and afterload: Roles in cardiac compensation and decompensation
J Physiol
PubMed 38686538 DOI 10.1113/JP284566
Li J, Sundnes J, Hou Y, Laasmaa M, Ruud M, Unger A, Kolstad TR, Frisk M, Norseng PA, Yang L, Setterberg IE, Alves ES, Kalakoutis M, Sejersted OM, Lanner JT, Linke WA, Lunde IG, de Tombe PP, Louch WE (2023)
Stretch Harmonizes Sarcomere Strain Across the Cardiomyocyte
Circ Res, 133 (3), 255-270
PubMed 37401464 DOI 10.1161/CIRCRESAHA.123.322588
Hou Y, Laasmaa M, Li J, Shen X, Manfra O, Norden ES, Le C, Zhang L, Sjaastad I, Jones PP, Soeller C, Louch WE (2023)
Live-cell photo-activated localization microscopy correlates nanoscale ryanodine receptor configuration to calcium sparks in cardiomyocytes
Nat Cardiovasc Res, 2 (3), 251-267
PubMed 38803363 DOI 10.1038/s44161-022-00199-2
Rypdal KB, Olav Melleby A, Robinson EL, Li J, Palmero S, Seifert DE, Martin D, Clark C, López B, Andreassen K, Dahl CP, Sjaastad I, Tønnessen T, Stokke MK, Louch WE, González A, Heymans S, Christensen G, Apte SS, Lunde IG (2022)
ADAMTSL3 knock-out mice develop cardiac dysfunction and dilatation with increased TGFβ signalling after pressure overload
Commun Biol, 5 (1), 1392
PubMed 36539599 DOI 10.1038/s42003-022-04361-1
Støle TP, Lunde M, Shen X, Martinsen M, Lunde PK, Li J, Lockwood F, Sjaastad I, Louch WE, Aronsen JM, Christensen G, Carlson CR (2022)
The female syndecan-4-/- heart has smaller cardiomyocytes, augmented insulin/pSer473-Akt/pSer9-GSK-3β signaling, and lowered SCOP, pThr308-Akt/Akt and GLUT4 levels
Front Cell Dev Biol, 10, 908126
PubMed 36092718 DOI 10.3389/fcell.2022.908126
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